Abstract Increasing proof implicates altered mTORC1 signaling cascades from the pathophysiology of melancholy, suggesting that direct modulation of mTORC1 signaling might offer novel therapeutic opportunity. In this challenge from the JCI, Kato and colleagues claimed that administration of NV-5138, a not long ago developed synthetic leucine analog, provides a rapid https://chesterl318bfk1.dm-blog.com/profile