Max in WT synaptoneurosomes, suggesting that Src signaling could be downregulated in KI synapses. However, our power to rescue SERT function in KI midbrain synaptoneurosomes via the inhibition of FAK signifies elevated FAK signaling downstream in the Pro32Pro33 mutant, as confirmed by increased pFAK localization in five-HT synapses. Our facts https://margareti319ite0.blogpixi.com/profile