Preliminary studies reveal conolidine may inhibit distinct ion channels, decreasing neuronal excitability and limiting soreness signals. This mechanism is especially appropriate in neuropathic ache, where by irregular signaling will cause persistent pain. Also, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to suffering perception. Computational docking... https://jeffg035hxe3.bloggip.com/profile